STAT1 Cancer Research Results

STAT1, Signal transducer and activator of transcription 1: Click to Expand ⟱
Source:
Type:
STAT1 (Signal Transducer and Activator of Transcription 1) is a transcription factor that plays a crucial role in various cellular processes, including immune response, cell growth, and apoptosis.
Unregulated in some cancers.
High STAT1 expression has been associated with improved prognosis and reduced metastasis.
Low STAT1 expression has been linked to poor prognosis and reduced survival.


Scientific Papers found: Click to Expand⟱
5988- Chit,    Chitosan immunomodulation: insights into mechanisms of action on immune cells and signaling pathways
- Review, Var, NA
DDS↑, *Cartilage↑, *Wound Healing↑, Imm↑, cGAS–STING↑, STAT1↑, NLRP3↑, *DCells↑, *IL10↓, *TGF-β1↓, *TNF-α↓, IL1β↓, ROS↑,

Showing Research Papers: 1 to 1 of 1

* indicates research on normal cells as opposed to diseased cells
Total Research Paper Matches: 1

Pathway results for Effect on Cancer / Diseased Cells:


Redox & Oxidative Stress

ROS↑, 1,  

Proliferation, Differentiation & Cell State

STAT1↑, 1,  

Immune & Inflammatory Signaling

IL1β↓, 1,   Imm↑, 1,  

Cellular Microenvironment

cGAS–STING↑, 1,  

Protein Aggregation

NLRP3↑, 1,  

Drug Metabolism & Resistance

DDS↑, 1,  
Total Targets: 7

Pathway results for Effect on Normal Cells:


Migration

Cartilage↑, 1,   TGF-β1↓, 1,  

Immune & Inflammatory Signaling

DCells↑, 1,   IL10↓, 1,   TNF-α↓, 1,  

Functional Outcomes

Wound Healing↑, 1,  
Total Targets: 6

Scientific Paper Hit Count for: STAT1, Signal transducer and activator of transcription 1
Query results interpretion may depend on "conditions" listed in the research papers.
Such Conditions may include : 
  -low or high Dose
  -format for product, such as nano of lipid formations
  -different cell line effects
  -synergies with other products 
  -if effect was for normal or cancerous cells
Filter Conditions: Pro/AntiFlg:%  IllCat:%  CanType:%  Cells:%  prod#:%  Target#:370  State#:%  Dir#:2
wNotes=0 sortOrder:rid,rpid

 

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