memory Cancer Research Results
memory, memory: Click to Expand ⟱
Scientific Papers found: Click to Expand⟱
*ROS↓, carnosine may be potentially beneficial in the treatment of AD because of its free-radical scavenger and metal chelating properties.
*IronCh↑, Carnosine chelates intracellular Zn2+
*Aβ↓, strong reduction in the hippocampal intraneuronal accumulation of Aβ
*AntiAge↑, Carnosine also exerts anti-aging activities by neutralizing injurious glycated proteins and aldehydic products of lipids peroxydation
*lipid-P↓,
*cognitive↑, We observed a positive trend toward a better cognitive performance as indicated by the decreased latency to find the platform
*memory∅, Carnosine supplementation was not able to completely rescue long-term memory deficits in treated 3xTg-AD mice.
*memory↑, Extensively used as food supplements, they have been shown to represent an effective strategy for boosting memory and enhancing cognitive function. Numerous clinical reports suggest that GPC can improve memory and attention in patients with Alzheimer
*cognitive↑,
*Dose↝, GPC is considered one of the most used sources of choline due to its high choline content (41% of choline by weight) and its ability to cross the blood-brain barrier.
*BBB↑,
*cardioP↑, Choline plays a protective role in the heart and may be a promising candidate to improve doxorubicin-induced cardiotoxicity via vagal activity and Nrf2/HO-1 pathway
*ROS↓, by inhibiting the ROS-mediated Ca2+/calmodulin-dependent protein kinase II pathway
*TMAO↑, In contrast, a choline‐ or carnitine‐rich diet was reported to promote atherosclerosis in mice as it increased the formation of TMAO produced by gut microbiota-related metabolite of choline
*memory∅, Choline supplements in the form of lecithin and choline chloride did not significantly improve memory performance in humans although some papers have reported positive outcomes in cognitive function of animal models
*eff↑, However, other choline supplements such as citicoline, choline bitartrate, and GPC appear to be very promising in the treatment of elderly patients suffering from dementia (49, 52, 54, 157, 158).
*OS↑, mice treated with HC exhibited significant delays in spontaneous early mortality at 70%–90% mice survival in the longevity study
*toxicity↓, suggesting that chronic exposure to HC does not produce toxicity at the given dose
*AST∅, (AST) and ALT markers of liver damage were not altered
*ALAT∅,
*Strength↑, Remarkably, HC produced an increase in wire hang performance
*memory∅, HC did not produce remarkable effects in neurocognitive health and muscle strength in HFD‐fed mice
*other↑, HC promotes a rich energetic status in the liver
*other↑, HC potentiates muscle regeneration in vivo
*other↑, HC potentiates muscle regeneration in vivo
*APP∅, No difference in hippocampal expression of APP-695, Aβ(1−42), S100b and GFAP proteins or in the pTau/Tau ratio was observed between sham- and ELF-MF-exposed aged mice
*Aβ∅,
*Inflam∅, ELF-MFs does not aggravate aging and associated neuroinflammation, or promote pathological pathways involved in the initiation of AD
*memory∅, However, sham- and ELF-MF-exposed aged mice were equally able to discriminate the newly accessible arm during the test phase, which resulted in a spatial recognition memory performance above chance level
*Weight∅, body weight of rats showed no difference compared with the control group.
*memory∅, application of ELF-EMF did not induce any cognitive and memory impairment compared with the sham-exposure group.
*cognitive∅,
*Aβ∅, Aβ showed no significant change between the two groups,
Showing Research Papers: 1 to 5 of 5
* indicates research on normal cells as opposed to diseased cells
Total Research Paper Matches: 5
Pathway results for Effect on Cancer / Diseased Cells:
Total Targets: 0
Pathway results for Effect on Normal Cells:
NA, unassigned ⓘ
TMAO↑, 1,
Redox & Oxidative Stress ⓘ
lipid-P↓, 1, ROS↓, 2,
Metal & Cofactor Biology ⓘ
IronCh↑, 1,
Core Metabolism/Glycolysis ⓘ
ALAT∅, 1,
Transcription & Epigenetics ⓘ
other↑, 3,
Migration ⓘ
APP∅, 1,
Barriers & Transport ⓘ
BBB↑, 1,
Immune & Inflammatory Signaling ⓘ
Inflam∅, 1,
Protein Aggregation ⓘ
Aβ↓, 1, Aβ∅, 2,
Drug Metabolism & Resistance ⓘ
Dose↝, 1, eff↑, 1,
Clinical Biomarkers ⓘ
ALAT∅, 1, AST∅, 1,
Functional Outcomes ⓘ
AntiAge↑, 1, cardioP↑, 1, cognitive↑, 2, cognitive∅, 1, memory↑, 1, memory∅, 5, OS↑, 1, Strength↑, 1, toxicity↓, 1, Weight∅, 1,
Total Targets: 25
Scientific Paper Hit Count for: memory, memory
Query results interpretion may depend on "conditions" listed in the research papers.
Such Conditions may include :
-low or high Dose
-format for product, such as nano of lipid formations
-different cell line effects
-synergies with other products
-if effect was for normal or cancerous cells
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